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10.1016/S1474-4422(17)30371-X 105 MaasA

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The compounds operate through distinct, non-overlapping mechanisms (GLP-1/GIP receptor agonism versus NNMT enzyme inhibition) with no documented receptor competition or adverse pharmacokinetic interactions

While GHK-Cu treatment alone did not cause any visible histological changes or toxicity, pretreatment with GHK-Cu markedly attenuated these pathological alterations, resulting in a significantly reduced lung injury score compared to the LPS-only group